Abstract Library

Welcome to the open-access search for all ENETS abstracts presented at the Annual ENETS Conferences.

Everyone can browse the library to find basic information on abstracts. To get full access to each entry, you will be asked to log in to your myENETS account.

 

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Participants of the 2025 ENETS Conference enjoy full access to the 2025 conference digital resources through myENETS: the abstract booklet, e-posters and videos, slide decks of talks, the poster carousel, and more.

ENETS Abstract Search

#2009 Retrospective Epidemiologic Study of Medullary Cancer of Thyroid Gland in Hospital – Based Registry in Moscow Cancer Centre

Introduction: Medullary cancer of thyroid gland comes from calcitonin secreting C-cells.

Conference: 15th Annual ENETSConcerence (2018)

Presenting Author:

Authors: Komarova L, Tabolinovskya T, Pirogova N, Machaladze Z,

Keywords: registry, NETs,

#631 Genotype – Phenotype Correlations and Timing of Prophylactic Thyroidectomy in Patients with Familial Medullary Thyroid Carcinoma

Introduction: Recommendations on the timing of prophylactic thyroidectomy (PTTE) in patients with familial medullary carcinoma (FMTC) are based on classification of RET mutations into four risk levels (ATA 2009).

Conference: 10th Annual ENETSConcerence (2013)

Presenting Author: Podoba J

Authors: Podoba J, Podobová M, Grigerová M, Weismanová E, Závodná K,

Keywords: familial medullary carcinoma, prophylactic thyroidectomy, timing,

#333 Sporadic Hypercalcitoninemia: Thyroid Medullary carcinoma, Autoimmune Thyroiditis or GLP-1 Agonists’ Effect?

Introduction: Glucagon-like peptide 1 (GLP-1) receptor agonists’ stimulated C-cell hyperplasia and tumors in rodents, but not in humans. Calcitonin also increased in autoimmune thyroid disease.

Conference: 8th Annual ENETSConcerence (2011)

Presenting Author:

Authors: Trifanescu R, Gherlan I, Carsote M, Poiana C,

Keywords: calcitonin, medullary thyroid carcinoma, GLP-1 agonist, autoimmune thyroiditis,

#276 Mammalian Target of Rapamycin Pathway Activation is Associated with RET Mutation Status in Medullary Thyroid Carcinoma

Introduction: The genetic pathways involved in medullary thyroid carcinoma (MTC), except for RET mutations, are largely unknown, as well as the detailed expression mapping of intracellular signalling transducers.

Conference: 8th Annual ENETSConcerence (2011)

Presenting Author:

Authors: Rapa I, Saggiorato E, Giachino D, Palestini N, Orlandi F,

Keywords: thyroid, medullary carcinoma, RET, mTOR, immunohistochemistry,