Abstract Library

#2229 The NETwork! Registry Suggests Increasing Incidence in a National Study of Neuroendocrine Cancer in New Zealand (NZ)

Introduction: NETwork! is a clinical and translational research program, including a retrospective epidemiological study of NETs in NZ.

Conference: 15th Annual ENETSConcerence (2018)

Presenting Author: Parker K

Authors: Parker K, Coats E, Woodhouse B, Knowlton N, Print C,

Keywords: neuroendocrine, incidence, epidemiology, registry,

#2226 Approaching NETs from Many Angles: A Medical Research Paradigm to Challenge New Zealand’s Research Traditions

Introduction: NETwork! is a multidisciplinary clinical and translational research program investigating NETs from several perspectives.

Conference: 15th Annual ENETSConcerence (2018)

Presenting Author: Parker K

Authors: Parker K, Blenkiron C, Print C, Findlay M, Lawrence B,

Keywords: neuroendocrine, translational, multidisciplinary,

#1694 Plasma Protein Fingerprinting for the Diagnosis of Small Intestinal Neuroendocrine Tumors (siNETs)

Introduction: siNETs are notoriously difficult to diagnose, especially in an early stage.

Conference: 14th Annual ENETSConcerence (2017)

Presenting Author: Kjellman M

Authors: Kjellman M, Knigge U, Welin S, Grønbæk H, This-Evensen E,

Keywords: NET biomarker diagnosis,

#1551 Establishment and Characterization of Three GEP-NEC Cell Lines for In Vitro and In Vivo Studies

Introduction: To date chemotherapeutic concepts applying cisplatin/etoposide for the treatment of highly aggressive GEP-NEC do not provide convincing results. Thus, there is an urgent need for NEC cell line models that provide a reliable experimental system in identifying novel therapeutic targets.

Conference: 13th Annual ENETSConcerence (2016)

Presenting Author: Krieg A

Authors: Krieg A, Riemer J, Dizdar L, Mersch S, Anlauf M,

Keywords: GEP-NEC,

#527 Pheochromocytoma Molecular Analysis After Maternal Transmission of SDHD Mutation Elucidates Mechanism of Parent-of-Origin Effect

Introduction: In SDHD mutation families, paragangliomas and pheochromocytomas usually occur only after paternal transmission of the mutation. This important but unexplained parent-of-origin effect is not due to imprinting of the SDHD gene itself (as was initially suspected) since SDHD is biallelically expressed.

Conference: 9th Annual ENETSConcerence (2012)

Presenting Author:

Authors: Tobias E, Yeap P, Mavraki E, Fletcher A, Freel M,

Keywords: SDHD, imprinting, paraganglioma,